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Sample Argumentative Essay on Annotated Bibliography

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Sample Argumentative Essay on Annotated Bibliography

Boulet, L. P., Lemière, C., Archambault, F., Carrier, G., Descary, M. C., & Deschesnes, F. (2006). Smoking and asthma: clinical and radiologic features, lung function, and airway inflammation. CHEST Journal, 129(3), 661-668.
The article is about the relationship between smoking and asthma regarding clinical and radiological features, airway inflammation, and lung function. The research question of the article is whether smoking brings changes in lung function and structure resulting to airway inflammation. The results gave data proving that smoking affects asthmatic airways in the lungs. The participants of the study were 18 Asthma patients taken from the Laval Hospital. Data used for the research was taken from Laval Hospital Asthma Clinics. The article will build the research paper to be written by providing information that links smoking and asthma.
Chaudhuri, R., Livingston, E., McMahon, A. D., Lafferty, J., Fraser, I., Spears, M. &Thomson,N. C. (2006). Effects of smoking cessation on lung function and airway inflammation in smokers with asthma. American journal of respiratory and critical care medicine, 174(2), 127-133.
The article spells out how smoking cessation affects lung function and air inflammation in smokers with an asthma history. The research question of the article is whether smoking cessation has any effects on the lung function and air inflammation of Asthmatic smokers. The researchers found out that participants that stopped smoking improved in terms of lung function and sputum neutrophil count.The participants of the study were32 asthmatic smokers. The researchers sourced their data from the Department of Respiratory Medicine, University of Glasgow. The article will contribute more information about smoking and asthma to the research paper to be written.
Hamilton, M., Wolf, J. L., Rusk, J., Beard, S. E., Clark, G. M., Witt, K., & Cagnoni, P. J. (2006). Effects of smoking on the pharmacokinetics of erlotinib. Clinical Cancer Research, 12(7), 2166-2171.
The article explains the effects of smoking on the pharmacokinetics of the Erlotinib. The purpose of the article is to make a comparison between smokers and non-smokers after taking Erlotinib. The research question is whether Erlotinib has similar effect on smokers and non-smokers. The researchers found out that smokers received less exposure to Erlotinib than non-smokers. The author concluded that the pharmacokinetics of Erlotinib in smokers is different from non-smokers. The research design involved a single-center open-label pharmacokinetic. The participants were healthy males. The researchers sourced their data from the U.S Department of Health and Human Services. They found out that reduced Erlotinib exposure in non-small cell lung cancer patients is influenced by their smoking status.
Hughes, A. N., O’Brien, M. E., Petty, W. J., Chick, J. B., Rankin, E., Woll, P. J., … & Price, A. (2009). Overcoming CYP1A1/1A2 mediated induction of metabolism by escalating erlotinib dose in current smokers. Journal of Clinical Oncology, 27(8), 1220-1226.
The article is about overcoming induction Mediated by CYP1A1/1A2 on increasing doses of Erlotinib cancer patients who smoke. The purpose of the article is to find out the maximum dose of erlotinib that cancer patients who smoke can tolerate in their bodies. The research question of the article is whether there are dose amounts of Erlotinib, which cancer patients that smoke can tolerate. The researchers found out that maximum eltotinib dosage that cancerpatients who smoke can tolerate is 300g. The participants were 22 cancer patients who smoke. The search design used was the random sampling methods. The data used was provided by the American Society of Clinical Oncology. The article is useful to the research paper as it provides information about the effect of smoking on cancer patients.
Lazarus, S. C., Chinchilli, V. M., Rollings, N. J., Boushey, H. A., Cherniack, R., Craig, T. J., … & Israel, E. (2007). Smoking affects response to inhaled corticosteroids or leukotriene receptor antagonists in asthma. American journal of respiratory and critical care medicine, 175(8), 783-790.
The article is about the effect of smoking on the response of inhaled leukotrine in asthmatic people. The purpose of the article is to evaluate the effect of smoke on Leukotrine response in asthmatic patients. The research question is whether smoking affects the therapy of asthma. The researchers found out that the response of corticosteroids in asthmatic patients is attenuated, thus requiring standard therapy. The participants of the study were 44 non-smokers and 39 smokers suffering from mild and chronic asthmas. The researchers obtained data from the American Thoracic Society. The article builds the research as it gives information on the effect of smoking on the treatment of diseases.

References
Boulet, L. P., Lemière, C., Archambault, F., Carrier, G., Descary, M. C., & Deschesnes, F. (2006). Smoking and asthma: clinical and radiologic features, lung function, and airway inflammation. CHEST Journal, 129(3), 661-668.
Chaudhuri, R., Livingston, E., McMahon, A. D., Lafferty, J., Fraser, I., Spears, M. & Thomson, N. C. (2006). Effects of smoking cessation on lung function and airway inflammation in smokers with asthma. American journal of respiratory and critical care medicine, 174(2), 127-133.
Hamilton, M., Wolf, J. L., Rusk, J., Beard, S. E., Clark, G. M., Witt, K., & Cagnoni, P. J. (2006). Effects of smoking on the pharmacokinetics of erlotinib. Clinical Cancer Research, 12(7), 2166-2171.
Hughes, A. N., O’Brien, M. E., Petty, W. J., Chick, J. B., Rankin, E., Woll, P. J., … & Price, A. (2009). Overcoming CYP1A1/1A2 mediated induction of metabolism by escalating erlotinib dose in current smokers. Journal of Clinical Oncology, 27(8), 1220-1226.
Lazarus, S. C., Chinchilli, V. M., Rollings,

Boulet, L. P., Lemière, C., Archambault, F., Carrier, G., Descary, M. C., & Deschesnes, F. (2006). Smoking and asthma: clinical and radiologic features, lung function, and airway inflammation. CHEST Journal, 129(3), 661-668.

The article is about the relationship between smoking and asthma regarding clinical and radiological features, airway inflammation, and lung function. The research question of the article is whether smoking brings changes in lung function and structure resulting to airway inflammation. The results gave data proving that smoking affects asthmatic airways in the lungs. The participants of the study were 18 Asthma patients taken from the Laval Hospital. Data used for the research was taken from Laval Hospital Asthma Clinics. The article will build the research paper to be written by providing information that links smoking and asthma.

Chaudhuri, R., Livingston, E., McMahon, A. D., Lafferty, J., Fraser, I., Spears, M. &Thomson,N. C. (2006). Effects of smoking cessation on lung function and airway inflammation in smokers with asthma. American journal of respiratory and critical care medicine, 174(2), 127-133.

The article spells out how smoking cessation affects lung function and air inflammation in smokers with an asthma history. The research question of the article is whether smoking cessation has any effects on the lung function and air inflammation of Asthmatic smokers. The researchers found out that participants that stopped smoking improved in terms of lung function and sputum neutrophil count.The participants of the study were32 asthmatic smokers. The researchers sourced their data from the Department of Respiratory Medicine, University of Glasgow. The article will contribute more information about smoking and asthma to the research paper to be written.

Hamilton, M., Wolf, J. L., Rusk, J., Beard, S. E., Clark, G. M., Witt, K., & Cagnoni, P. J. (2006). Effects of smoking on the pharmacokinetics of erlotinib. Clinical Cancer Research, 12(7), 2166-2171.

The article explains the effects of smoking on the pharmacokinetics of the Erlotinib. The purpose of the article is to make a comparison between smokers and non-smokers after taking Erlotinib. The research question is whether Erlotinib has similar effect on smokers and non-smokers. The researchers found out that smokers received less exposure to Erlotinib than non-smokers. The author concluded that the pharmacokinetics of Erlotinib in smokers is different from non-smokers. The research design involved a single-center open-label pharmacokinetic. The participants were healthy males. The researchers sourced their data from the U.S Department of Health and Human Services. They found out that reduced Erlotinib exposure in non-small cell lung cancer patients is influenced by their smoking status.

Hughes, A. N., O’Brien, M. E., Petty, W. J., Chick, J. B., Rankin, E., Woll, P. J., … & Price, A. (2009). Overcoming CYP1A1/1A2 mediated induction of metabolism by escalating erlotinib dose in current smokers. Journal of Clinical Oncology, 27(8), 1220-1226.

The article is about overcoming induction Mediated by CYP1A1/1A2 on increasing doses of Erlotinib cancer patients who smoke. The purpose of the article is to find out the maximum dose of erlotinib that cancer patients who smoke can tolerate in their bodies. The research question of the article is whether there are dose amounts of Erlotinib, which cancer patients that smoke can tolerate. The researchers found out that maximum eltotinib dosage that cancerpatients who smoke can tolerate is 300g. The participants were 22 cancer patients who smoke. The search design used was the random sampling methods. The data used was provided by the American Society of Clinical Oncology. The article is useful to the research paper as it provides information about the effect of smoking on cancer patients.
Lazarus, S. C., Chinchilli, V. M., Rollings, N. J., Boushey, H. A., Cherniack, R., Craig, T. J., … & Israel, E. (2007). Smoking affects response to inhaled corticosteroids or leukotriene receptor antagonists in asthma. American journal of respiratory and critical care medicine, 175(8), 783-790.

The article is about the effect of smoking on the response of inhaled leukotrine in asthmatic people. The purpose of the article is to evaluate the effect of smoke on Leukotrine response in asthmatic patients. The research question is whether smoking affects the therapy of asthma. The researchers found out that the response of corticosteroids in asthmatic patients is attenuated, thus requiring standard therapy. The participants of the study were 44 non-smokers and 39 smokers suffering from mild and chronic asthmas. The researchers obtained data from the American Thoracic Society. The article builds the research as it gives information on the effect of smoking on the treatment of diseases.

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

References

Boulet, L. P., Lemière, C., Archambault, F., Carrier, G., Descary, M. C., & Deschesnes, F. (2006). Smoking and asthma: clinical and radiologic features, lung function, and airway inflammation. CHEST Journal, 129(3), 661-668.

Chaudhuri, R., Livingston, E., McMahon, A. D., Lafferty, J., Fraser, I., Spears, M. & Thomson, N. C. (2006). Effects of smoking cessation on lung function and airway inflammation in smokers with asthma. American journal of respiratory and critical care medicine, 174(2), 127-133.

Hamilton, M., Wolf, J. L., Rusk, J., Beard, S. E., Clark, G. M., Witt, K., & Cagnoni, P. J. (2006). Effects of smoking on the pharmacokinetics of erlotinib. Clinical Cancer Research, 12(7), 2166-2171.

Hughes, A. N., O’Brien, M. E., Petty, W. J., Chick, J. B., Rankin, E., Woll, P. J., … & Price, A. (2009). Overcoming CYP1A1/1A2 mediated induction of metabolism by escalating erlotinib dose in current smokers. Journal of Clinical Oncology, 27(8), 1220-1226.

Lazarus, S. C., Chinchilli, V. M., Rollings, N. J., Boushey, H. A., Cherniack, R., Craig, T. J., … & Israel, E. (2007). Smoking affects response to inhaled corticosteroids or leukotriene receptor antagonists in asthma. American journal of respiratory and critical care medicine, 175(8), 783-790.

N. J., Boushey, H. A., Cherniack, R., Craig, T. J., … & Israel, E. (2007). Smoking affects response to inhaled corticosteroids or leukotriene receptor antagonists in asthma. American journal of respiratory and critical care medicine, 175(8), 783-790.

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